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【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103室

這一成果發(fā)表在美國《科學轉化醫(yī)學》雜志上。陳萬軍說，他們利用此技術對罹患多發(fā)性硬化癥與I型糖尿病的小鼠進行治療，讓病鼠取得了長期緩解，接下來他們還將探索此技術在類風濕性關節(jié)炎治療與異體抗原抗體排斥治療等方面的應用前景。
他說，如果動物實驗成功，“我們將和臨床醫(yī)生合作把這個技術用在自體免疫疾病病人的治療上。盡管仍有很多困難和問題需要解決，但我們有信心zui終會造福病人。我們不但要和美國醫(yī)生合作，也希望有機會和中國醫(yī)生合作”。
南安普敦大學科學家的一項研究發(fā)現(xiàn)，神經(jīng)發(fā)生(神經(jīng)再生)，成人大腦的自我修復機制，有助于保護神經(jīng)退行性疾病(如阿爾茨海默氏癥，朊病毒或帕金森氏)的大腦功能。
許多神經(jīng)退行性疾病發(fā)生后，大腦會逐漸退化和死亡，這往往被視為一種不可阻擋的，不可撤銷的過程。然而，大腦有一些自我修復的潛力，更新生活在齒狀回中的某些神經(jīng)元的量。齒狀回是一個簡單的皮層區(qū)域，控制學習和記憶的大腦較大功能系統(tǒng)海馬體的一部分。這個過程被稱為神經(jīng)發(fā)生。
雖然過去的神經(jīng)退行性疾病中已報道增加神經(jīng)發(fā)生，但其意義尚不清楚。現(xiàn)在，來自于南安普敦大學生物科學中心迭戈戈麥斯 - 尼古拉博士的一個研究小組，發(fā)現(xiàn)，增加齒狀回的神經(jīng)發(fā)生可部分抵消神經(jīng)元的損傷。

This result was published in the United States, "Science Translational Medicine" magazine. Chen Wanjun said that they use this technology to treat mice with multiple sclerosis and type I diabetes so as to achieve long-term relief of the disease. Next, they will explore the use of this technology in the treatment of rheumatoid arthritis with alloantibodies Exclusion treatment and other aspects of the application prospects.
He said that if the animal experiments were successful, "We will work with clinicians to apply this technology to the treatment of patients with autoimmune diseases, and although there are still many difficulties and problems to solve, we are confident that we will ultimay benefit the patients. Cooperation with American doctors also hope to have the opportunity to cooperate with Chinese doctors. "
A study led by scientists at the University of Southampton found that neurogenesis (neural regeneration), the self-repair mechanism of the adult brain, helps protect neurodegenerative diseases such as Alzheimer's disease, prion or Parkinson's disease Brain function.
After many neurodegenerative diseases occur, the brain gradually degenerates and dies, often as an irresistible and irrevocable process. However, the brain has some potential for self-healing, updating the amount of certain neurons that live in the dentate gyrus. The dentate gyrus is a simple cortical area that controls part of the hippocampus of the greater functional system of learning and memory of the brain. This process is called neurogenesis.
Although neurogenesis has been reported in neurodegenerative diseases in the past, its significance is not clear. Now, a team led by Dr Diego Gomez-Nicola, from the Center for Biological Sciences at the University of Southampton, found that increasing neurogenesis in the dentate gyrus partially offset neuronal damage.